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 Session 10: Mitochondria and Degenerative Diseases – II. Patients and Human Cell LinesMonday, 19. Sept. 10:45 Chair: Sergio Papa (Bari IT, papabchm@cimedoc.uniba.it)
Patrick Schrauwen (Maastricht NL, p.schrauwen@hb.unimaas.nl) 10:50-12:05 Oral presentations 10+5 min
10-01. 10:50-11:00 Patrick Schrauwen (Maastricht NL) Oxidative capacity, lipotoxicity and mitochondrial function in type 2 diabetes mellitus.
10-02. 11:05-11:15 Rolf Berge (Bergen NO) Dissipating excess energy in the liver is a potential treatment strategy for the metabolic syndrome.
10-03. 11:20-11:30 Wolfgang F Graier (Graz AT) Endothelial mitochondria exhibit an initial target of hyperglycemia.
10-04. 11:35-11:45 Sergio Papa (Bari IT) Complex I and physiopathology of mitochondrial oxygen metabolism.
10-05. 11:50-12:00 Josef Houstek (Prague CZ) Two components in pathogenic mechanism of mitochondrial ATPase deficiency: Energy deprivation and ROS production.
10-06. Poster E3 Werner JH Koopmanm (Nijmegen NL) Trolox restores aberrant mitochondrial morphology and improves assembly and activity of complex I in patients with an isolated deficiency.
10-07. Poster E4 Petr Pecina (Prague CZ) Cytochrome c oxidase assembly defects. [YIP]
10-08. Poster E5 Karl Johan Tronstad (Bergen NO) Mitochondrial alterations during apoptosis induced by a thia fatty acid in leukemia cells.
10-09. Poster E6 Roberto Scatena (Rome IT) Mitochondrial derangement induced by PPAR-ligands in HEP-G2 cell lines. Pathophysiological and pharmacotoxicological implications.
10-10. Poster E7 Hong Kyu Lee (Seoul KR) Cancer cell growth can be affected by different mitochondrial DNAs from subjects without cancer.
10-11. Poster E8 Denis Pierron (Bordeaux FR) Importance of the mitochondrial genetic background in the mtDNA mutations expression: Implication in mitochondrial diseases. [YIP]
10-12. Poster E9 Cenk Aral (Istanbul TR) A novel approach for rapid screening of mitochondrial D310 polymorphism. [YIP]
10-13. Poster E10 Flemming Dela (Copenhagen DK) Reduced skeletal muscle mitochondrial O2 flux capacity in type 2 diabetes.
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