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Difference between revisions of "Skladal 1994 BTK-268"

From Bioblast
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|year=1994
|year=1994
|journal=Innsbruck Univ Press
|journal=Innsbruck Univ Press
|mipnetlab=AT_Innsbruck_Gnaiger E
|mipnetlab=AT_Innsbruck_Gnaiger E, AT Innsbruck MitoCom
|discipline=Mitochondrial Physiology
|discipline=Mitochondrial Physiology
}}
}}
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|tissues=Skeletal muscle
|tissues=Skeletal muscle
|preparations=Permeabilized tissue
|preparations=Permeabilized tissue
|topics=Respiration; OXPHOS; ETS Capacity, Substrate; Glucose; TCA Cycle
|couplingstates=OXPHOS
|topics=Substrate; Glucose; TCA Cycle
|discipline=Mitochondrial Physiology
|discipline=Mitochondrial Physiology
}}
}}
* [[Tissue storage]]
* [[Tissue storage]]

Revision as of 14:30, 7 December 2012

Publications in the MiPMap
Skladal D, Sperl W, Schranzhofer R, Krismer M, Gnaiger E, Margreiter R, Gellerich FN (1994) Preservation of mitochondrial functions in human skeletal muscle during storage in high energy preservation solution (HEPS). In: What is Controlling Life? (Gnaiger E, Gellerich FN, Wyss M, eds) Modern Trends in BioThermoKinetics 3. Innsbruck Univ Press: 268-271.

Β» pdf

Skladal D, Sperl W, Schranzhofer R, Krismer M, Gnaiger E, Margreiter R, Gellerich FN (1994) Innsbruck Univ Press

Abstract:


β€’ O2k-Network Lab: AT_Innsbruck_Gnaiger E, AT Innsbruck MitoCom


Labels:

Stress:Ischemia-Reperfusion; Preservation"Ischemia-Reperfusion; Preservation" is not in the list (Cell death, Cryopreservation, Ischemia-reperfusion, Permeability transition, Oxidative stress;RONS, Temperature, Hypoxia, Mitochondrial disease) of allowed values for the "Stress" property.  Organism: Human  Tissue;cell: Skeletal muscle  Preparation: Permeabilized tissue 

Regulation: Substrate; Glucose; TCA Cycle"Substrate; Glucose; TCA Cycle" is not in the list (Aerobic glycolysis, ADP, ATP, ATP production, AMP, Calcium, Coupling efficiency;uncoupling, Cyt c, Flux control, Inhibitor, ...) of allowed values for the "Respiration and regulation" property.  Coupling state: OXPHOS 

HRR: Oxygraph-2k