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Difference between revisions of "User talk:Nirschl Lisa"

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==MitoEAGLE Dissemination==
==MitoEAGLE Disseminations==
 
 
===Publications===
===Publications===
::::* [https://www.tcd.ie/research/themes/cancer/publications/index.php?year=*&pub_type=INVITED_TALK| Invited Talks: Publications-Cancer; Trinity Research]
::::::1995-2018
::::* [https://www.sciencedirect.com/science/article/pii/S0891584918305185| The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells]
::::* [https://www.sciencedirect.com/science/article/pii/S0891584918305185| The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells]
::::::Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353
::::::Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353
==Publications found/ added link:==
::::* [https://www.nature.com/articles/pr2017250#acknowledgements| Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease]
::::::Westerlund, E., Marelsson, S. E., Ehinger, J. K., Sjövall, F., Morota, S., Frostner, E. Å, Oldfors, A., Darin, N., Lundgren, J., Hansson, M., Fellman, V., Elmér, E. (2017). Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease. Pediatric Research, 83(2), 455-465. doi:10.1038/pr.2017.250
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Biological+Implications+of+Differential+Expression+of+Mitochondrial-Shaping+Proteins+in+Parkinson%E2%80%99s+Disease| Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson's Disease, 2017]
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Biological+Implications+of+Differential+Expression+of+Mitochondrial-Shaping+Proteins+in+Parkinson%E2%80%99s+Disease| Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson's Disease, 2017]
::::::Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
::::::Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
::::* [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5460290| Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: electron flow through the Q-junction in permeabilized fibers]
::::* [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5460290| Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: electron flow through the Q-junction in permeabilized fibers]
::::::Lemieux, H., Blier, P. U., & Gnaiger, E. (2017). Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: Electron flow through the Q-junction in permeabilized fibers. Scientific Reports, 7(1). doi:10.1038/s41598-017-02789-8
::::* [https://www.biorxiv.org/content/early/2017/06/19/151480| Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal]
::::::Lemieux, H., Subarsky, P., Doblander, C., Wurm, M., Troppmair, J., & Gnaiger, E. (2017). Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal. doi:10.1101/151480
::::* [https://www.tandfonline.com/doi/full/10.1080/01480545.2016.1255953?scroll=top&needAccess=true| Acetaminophen toxicity in rat and mouse hepatocytes in vitro]
::::::Kučera, O., Endlicher, R., Rychtrmoc, D., Lotková, H., Sobotka, O., & Červinková, Z. (2016). Acetaminophen toxicity in rat and mouse hepatocytes in vitro. Drug and Chemical Toxicology, 40(4), 448-456. doi:10.1080/01480545.2016.1255953
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Effects+of+Epigallocatechin+Gallate+on+Tert-Butyl+Hydroperoxide-Induced+Mitochondrial+Dysfunction+in+Rat+Liver+Mitochondria+and+Hepatocytes| Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes]
::::::Mezera, V., Endlicher, R., Kucera, O., Sobotka, O., Drahota, Z., & Cervinkova, Z. (2016). Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes. Oxidative Medicine and Cellular Longevity, 2016, 1-8. doi:10.1155/2016/7573131

Latest revision as of 11:10, 18 January 2019

MitoEAGLE Disseminations

Publications

1995-2018
Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353





Publications found/ added link:

Westerlund, E., Marelsson, S. E., Ehinger, J. K., Sjövall, F., Morota, S., Frostner, E. Å, Oldfors, A., Darin, N., Lundgren, J., Hansson, M., Fellman, V., Elmér, E. (2017). Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease. Pediatric Research, 83(2), 455-465. doi:10.1038/pr.2017.250
Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
Lemieux, H., Blier, P. U., & Gnaiger, E. (2017). Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: Electron flow through the Q-junction in permeabilized fibers. Scientific Reports, 7(1). doi:10.1038/s41598-017-02789-8
Lemieux, H., Subarsky, P., Doblander, C., Wurm, M., Troppmair, J., & Gnaiger, E. (2017). Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal. doi:10.1101/151480
Kučera, O., Endlicher, R., Rychtrmoc, D., Lotková, H., Sobotka, O., & Červinková, Z. (2016). Acetaminophen toxicity in rat and mouse hepatocytes in vitro. Drug and Chemical Toxicology, 40(4), 448-456. doi:10.1080/01480545.2016.1255953
Mezera, V., Endlicher, R., Kucera, O., Sobotka, O., Drahota, Z., & Cervinkova, Z. (2016). Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes. Oxidative Medicine and Cellular Longevity, 2016, 1-8. doi:10.1155/2016/7573131