Cookies help us deliver our services. By using our services, you agree to our use of cookies. More information

Difference between revisions of "User talk:Nirschl Lisa"

From Bioblast
 
(20 intermediate revisions by the same user not shown)
Line 1: Line 1:
==MitoEAGLE Disseminations==
==MitoEAGLE Disseminations==


===Project Partners===
 
::::* [http://portal.research.lu.se/portal/en/projects/eu-fp-horizon-2020-cost-action-ca15203-mitoeagle(06fb61a9-eecf-49de-beeb-fea139bb6b28).html| EU FP Horizon 2020]
::::::09/2016-09/2020
::::* [https://trauma.lbg.ac.at/en/projects/mitoeagle| Ludwig Boltzmann Institute]
::::::since 2016
===Publications===
===Publications===
::::* [https://www.tcd.ie/research/themes/cancer/publications/index.php?year=*&pub_type=INVITED_TALK| Invited Talks: Publications-Cancer; Trinity Research]
::::* [https://www.tcd.ie/research/themes/cancer/publications/index.php?year=*&pub_type=INVITED_TALK| Invited Talks: Publications-Cancer; Trinity Research]
::::::1995-2018
::::::1995-2018
::::* [https://www.sciencedirect.com/science/article/pii/S0891584918305185| The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells]
::::* [https://www.sciencedirect.com/science/article/pii/S0891584918305185| The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells]
::::::Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353
::::::Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353
==Publications found/ added link:==
::::* [https://www.nature.com/articles/pr2017250#acknowledgements| Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease]
::::::Westerlund, E., Marelsson, S. E., Ehinger, J. K., Sjövall, F., Morota, S., Frostner, E. Å, Oldfors, A., Darin, N., Lundgren, J., Hansson, M., Fellman, V., Elmér, E. (2017). Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease. Pediatric Research, 83(2), 455-465. doi:10.1038/pr.2017.250
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Biological+Implications+of+Differential+Expression+of+Mitochondrial-Shaping+Proteins+in+Parkinson%E2%80%99s+Disease| Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson's Disease, 2017]
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Biological+Implications+of+Differential+Expression+of+Mitochondrial-Shaping+Proteins+in+Parkinson%E2%80%99s+Disease| Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson's Disease, 2017]
::::::Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
::::::Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
::::* [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5460290 | Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: electron flow through the Q-junction in permeabilized fibers]
 
::::* [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5460290| Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: electron flow through the Q-junction in permeabilized fibers]
::::::Lemieux, H., Blier, P. U., & Gnaiger, E. (2017). Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: Electron flow through the Q-junction in permeabilized fibers. Scientific Reports, 7(1). doi:10.1038/s41598-017-02789-8
::::::Lemieux, H., Blier, P. U., & Gnaiger, E. (2017). Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: Electron flow through the Q-junction in permeabilized fibers. Scientific Reports, 7(1). doi:10.1038/s41598-017-02789-8
::::* [https://www.biorxiv.org/content/early/2017/06/19/151480| Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal]
::::* [https://www.biorxiv.org/content/early/2017/06/19/151480| Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal]
::::::Lemieux, H., Subarsky, P., Doblander, C., Wurm, M., Troppmair, J., & Gnaiger, E. (2017). Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal. doi:10.1101/151480
::::::Lemieux, H., Subarsky, P., Doblander, C., Wurm, M., Troppmair, J., & Gnaiger, E. (2017). Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal. doi:10.1101/151480
::::* [https://www.tandfonline.com/doi/full/10.1080/01480545.2016.1255953?scroll=top&needAccess=true| Acetaminophen toxicity in rat and mouse hepatocytes in vitro]
::::* [https://www.tandfonline.com/doi/full/10.1080/01480545.2016.1255953?scroll=top&needAccess=true| Acetaminophen toxicity in rat and mouse hepatocytes in vitro]
::::::Kučera, O., Endlicher, R., Rychtrmoc, D., Lotková, H., Sobotka, O., & Červinková, Z. (2016). Acetaminophen toxicity in rat and mouse hepatocytes in vitro. Drug and Chemical Toxicology, 40(4), 448-456. doi:10.1080/01480545.2016.1255953
::::::Kučera, O., Endlicher, R., Rychtrmoc, D., Lotková, H., Sobotka, O., & Červinková, Z. (2016). Acetaminophen toxicity in rat and mouse hepatocytes in vitro. Drug and Chemical Toxicology, 40(4), 448-456. doi:10.1080/01480545.2016.1255953
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Effects+of+Epigallocatechin+Gallate+on+Tert-Butyl+Hydroperoxide-Induced+Mitochondrial+Dysfunction+in+Rat+Liver+Mitochondria+and+Hepatocytes| Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes]
::::* [https://www.ncbi.nlm.nih.gov/pubmed/?term=Effects+of+Epigallocatechin+Gallate+on+Tert-Butyl+Hydroperoxide-Induced+Mitochondrial+Dysfunction+in+Rat+Liver+Mitochondria+and+Hepatocytes| Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes]
::::::Mezera, V., Endlicher, R., Kucera, O., Sobotka, O., Drahota, Z., & Cervinkova, Z. (2016). Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes. Oxidative Medicine and Cellular Longevity, 2016, 1-8. doi:10.1155/2016/7573131
::::::Mezera, V., Endlicher, R., Kucera, O., Sobotka, O., Drahota, Z., & Cervinkova, Z. (2016). Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes. Oxidative Medicine and Cellular Longevity, 2016, 1-8. doi:10.1155/2016/7573131
===Social Media===
::::*[https://www.youtube.com/watch?v=KtVYLfJYJMI| Incontro della rete europea Mitoeagle -  - Meeting of the European MitoEAGLE Network]
::::::''Translation'': The first meeting of the European network (MitoEAGLE) for the study of Mitochondrial Bioenergetics was held in Verona from November 15th to 17th with researchers from all over Europe, who gathered to take stock of research and share data and study methods.
::::::''Erich Gnaiger'': "This network aims to bring together scientists and researchers working in the field of Mitochondrial Research to share data methodologies. One of the slogans that we coined on this occasion is “By sharing data we will be stronger”."
::::::''Elisa Calabria'': "Mitochondrial transcription may be one of the indexes that will be integrated as a health index - we are particularly interested in aging and fragility in aging and we think we can find some parameters that will allow us to predict the good trend of our aging rather than a state of fragility in mitochondrial respiration."
::::::''Federico Schena": "Starting from mitochondria seems to be a very far start but this is todays research method, it is a research that is molecular in its bases and then must necessarily become applied translational. Therefore, being within the COST project is fully consistent with the path of training and qualification of the research for Sport Sciences which, however, has been pursuing for years and which in these years is certainly also a considerable satisfaction. For us, the theme of aging and the relationship between physical activity and lifestyle aging is a theme that we have been following for a long time also in collaboration on the territory with the local municipality, hospital and region, but also with broad vision. This project helps us to be qualified from a scientific point of view but at the same time also to be fully at the service of the people who live in Verona, who find in the physical activity an instrument of well-being of quality of life."

Latest revision as of 11:10, 18 January 2019

MitoEAGLE Disseminations

Publications

1995-2018
Rodriguez-Hernández, A., Staňková, P., González, R., Rosa, Á J., Álamo-Martinez, J. M., Marin-Gómez, L. M., Sobotka, O., Kučera, O., Padilli, F.J., Červinková, Z., Muntané, J. (2018). The alteration of mitochondrial function is associated with the induction of endoplasmic reticulum stress autophagy and apoptosis by Sorafenib in liver cancer cells. Free Radical Biology and Medicine, 120, 107. doi:10.1016/j.freeradbiomed.2018.04.353





Publications found/ added link:

Westerlund, E., Marelsson, S. E., Ehinger, J. K., Sjövall, F., Morota, S., Frostner, E. Å, Oldfors, A., Darin, N., Lundgren, J., Hansson, M., Fellman, V., Elmér, E. (2017). Oxygen consumption in platelets as an adjunct diagnostic method for pediatric mitochondrial disease. Pediatric Research, 83(2), 455-465. doi:10.1038/pr.2017.250
Rocha, S., Freitas, A., Guimaraes, S., Vitorino, R., Aroso, M., & Gomez-Lazaro, M. (2017). Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson’s Disease. Antioxidants, 7(1). doi:10.3390/antiox7010001
Lemieux, H., Blier, P. U., & Gnaiger, E. (2017). Remodeling pathway control of mitochondrial respiratory capacity by temperature in mouse heart: Electron flow through the Q-junction in permeabilized fibers. Scientific Reports, 7(1). doi:10.1038/s41598-017-02789-8
Lemieux, H., Subarsky, P., Doblander, C., Wurm, M., Troppmair, J., & Gnaiger, E. (2017). Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal. doi:10.1101/151480
Kučera, O., Endlicher, R., Rychtrmoc, D., Lotková, H., Sobotka, O., & Červinková, Z. (2016). Acetaminophen toxicity in rat and mouse hepatocytes in vitro. Drug and Chemical Toxicology, 40(4), 448-456. doi:10.1080/01480545.2016.1255953
Mezera, V., Endlicher, R., Kucera, O., Sobotka, O., Drahota, Z., & Cervinkova, Z. (2016). Effects of Epigallocatechin Gallate on Tert-Butyl Hydroperoxide-Induced Mitochondrial Dysfunction in Rat Liver Mitochondria and Hepatocytes. Oxidative Medicine and Cellular Longevity, 2016, 1-8. doi:10.1155/2016/7573131