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Difference between revisions of "Willems 2015 Cell Metab"

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{{Publication
|title=Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ (2015) Redox homeostasis and mitochondrial dynamics. Cell Metab 22:207-18. ย 
|title=Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ (2015) Redox homeostasis and mitochondrial dynamics. Cell Metab 22:207-18.
|info=[http://www.ncbi.nlm.nih.gov/pubmed/26166745 PMID: 26166745 Open Access]
|info=[http://www.ncbi.nlm.nih.gov/pubmed/26166745 PMID: 26166745 Open Access]
|authors=Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ
|authors=Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ
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Copyright ยฉ 2015 Elsevier Inc. All rights reserved.
Copyright ยฉ 2015 Elsevier Inc. All rights reserved.
|mipnetlab=FR Bordeaux Rossignol R
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Revision as of 15:31, 26 March 2018

Publications in the MiPMap
Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ (2015) Redox homeostasis and mitochondrial dynamics. Cell Metab 22:207-18.

ยป PMID: 26166745 Open Access

Willems PH, Rossignol R, Dieteren CE, Murphy MP, Koopman WJ (2015) Cell Metab

Abstract: Within living cells, mitochondria are considered relevant sources of reactive oxygen species (ROS) and are exposed to reactive nitrogen species (RNS). During the last decade, accumulating evidence suggests that mitochondrial (dys)function, ROS/RNS levels, and aberrations in mitochondrial morphology are interconnected, albeit in a cell- and context-dependent manner. Here it is hypothesized that ROS and RNS are involved in the short-term regulation of mitochondrial morphology and function via non-transcriptional pathways. We review the evidence for such a mechanism and propose that it allows homeostatic control of mitochondrial function and morphology by redox signaling.

Copyright ยฉ 2015 Elsevier Inc. All rights reserved.


โ€ข O2k-Network Lab: FR Bordeaux Rossignol R


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Stress:Oxidative stress;RONS