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Petit Patrice X

From Bioblast


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BEC 2020.1 Mitochondrial physiology
       
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COST Action CA15203 (2016-2021): MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping


Petit Patrice X


MitoPedia topics: EAGLE 

COST: Member

COST WG2: WG2

COST WG4: WG4


Name Petit Patrice X, Dr.,

Research Director CNRS,

Group leader "mitochondria, apoptosis and autophagy signaling"

Institution
Patrice Petit
Institute of Neuroscience,

Paris Descartes University, FR

Address 45, rue des Saints-Pรจres, 75006
City Paris
State/Province
Country France
Email [email protected]
Weblink
O2k-Network Lab


Labels:



Publications

 PublishedReference
BEC 2020.1 doi10.26124bec2020-0001.v12020Gnaiger E et al โ€• MitoEAGLE Task Group (2020) Mitochondrial physiology. Bioenerg Commun 2020.1. https://doi.org/10.26124/bec:2020-0001.v1

Abstracts

 PublishedReference
Rainey 2019 MiP20192019
Nathan Rainey
Mitochondria are not the primary target of curcumin-induced autophagy and apoptosis.
Petit 2019 MiP20192019
Patrice Petit
No abstract, Co-autor of Rainey 2019 MiP2019.
Petit 2018 MiP20182018
Patrice Petit
Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells with dirupted mitochondrial homeostasis (Barth syndrome cell model).
Petit 2017 MiP20172017
Patrice Petit
Barth syndrome cardiolipin alterations linked to tafazzin mutations lead to apoptosis and mitophygy alterations.
Petit 2013 Abstract MiP20132013Petit PX (2013) The cardiolipin defect linked to a tafazzin mutation in Barth syndrome disturbs mitochondrial metabolism, alters apoptosis and autophagic fluxes but does not affect cell cycle progression. Mitochondr Physiol Network 18.08.



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