Difference between revisions of "Liu 2018 Atherosclerosis Suppl"
From Bioblast
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|title=Liu D (2018) Enhanced Trpc3-mediated mitochondrial respiratory dysfunction by inhibition of mitochondria-hexokinase in monocytes from hypertension rats. Atherosclerosis Suppl. | |title=Liu D (2018) Enhanced Trpc3-mediated mitochondrial respiratory dysfunction by inhibition of mitochondria-hexokinase in monocytes from hypertension rats. Atherosclerosis Suppl. | ||
|info=[https://www.atherosclerosis-supplements.com/article/S1567-5688(18)30342-8/abstract Atherosclerosis] | |info=[https://www.atherosclerosis-supplements.com/article/S1567-5688(18)30342-8/abstract Atherosclerosis] | ||
|authors=Liu | |authors=Liu Daoyan | ||
|year=2018 | |year=2018 | ||
|event=Atherosclerosis Suppl | |event=Atherosclerosis Suppl | ||
Revision as of 18:52, 29 February 2020
| Liu D (2018) Enhanced Trpc3-mediated mitochondrial respiratory dysfunction by inhibition of mitochondria-hexokinase in monocytes from hypertension rats. Atherosclerosis Suppl. |
Link: Atherosclerosis
Liu Daoyan (2018)
Event: Atherosclerosis Suppl
Inhibition of transient receptor potential channel, canonical type 3 (TRPC3) attenuates monocytes and vasculature calcium influx in spontaneously hypertensive rats (SHRs). However, it remains elusive whether mitochondrial TRPC3 participates in increasing mitochondrial calcium homeostasis and ROS production in monocytes from hypertension.
• Bioblast editor: Plangger M, Kandolf G
• O2k-Network Lab: CN Chongqing Zhu Z
Labels: MiParea: Respiration, nDNA;cell genetics
Organism: Rat
Tissue;cell: Blood cells
Preparation: Intact cells
Coupling state: ROUTINE, OXPHOS, ET
Pathway: N, NS
HRR: Oxygraph-2k
Affiliations
- Daping Hospital, Third Military Medical Univ, Chongqing, China
