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O2k repair + ('''O2k repair''' of defective hardware may … '''O2k repair''' of defective hardware may require replacement of spare parts. Some electronic or mechanical defects may be solved only by repair of the O2k in the electronics workshop of Oroboros Instruments, ''e.g.'', a defective Peltier unit (temperature control).ective Peltier unit (temperature control).)
O2k status line + ('''O2k status line''' is found above the [[O2k signal line]] … '''O2k status line''' is found above the [[O2k signal line]]. It contains information about the chamber label, O2 calibration, amperometric calibration, potentiometric calibration, the [[block temperature]], the [[illumination]] in chambers, the TIP2k status and the [[Automatic pan]].[[Automatic pan]].)
O2k + ('''O2k''' - [[Oroboros O2k]]: the modular system for [[high-resolution respirometry]].)
O2k-Amperometric OroboPOS Twin-Channel + ('''O2k-Amperometric OroboPOS Twin-Channel' … '''O2k-Amperometric OroboPOS Twin-Channel''': Two-channel variable polarization voltage; current/voltage converter for the polarographic oxygen sensor (POS); amplifyer with digital gain settings (1x, 2x, 4x, 8x); A/D converter; output in the range -10 V to 10 V. Integral component of the [[O2k-Main Unit]].[[O2k-Main Unit]].)
O2k-Barometric Pressure Transducer + ('''O2k-Barometric Pressure Transducer''', … '''O2k-Barometric Pressure Transducer''', A/D converter and digital output to DatLab for continuous recording of [[barometric pressure]] [kPa or mmHg], integrated into the air calibration of the POS ([[MiPNet06.03 POS-calibration-SOP]]). Integral component of the [[O2k-Main Unit]]. The warranty on the accuracy of the signal obtained from the O2k-Barometric Pressure Transducer expires within three years.ure Transducer expires within three years.)
O2k-Electromagnetic Stirrer Twin-Control + ('''O2k-Electromagnetic Stirrer Twin-Contro … '''O2k-Electromagnetic Stirrer Twin-Control''' for smooth rotation of the [[Stirrer-Bar\white PVDF\15x6 mm|stirrer bars]] in the two [[O2k-chamber]]s; with slow-start function to prevent decoupling of the stirrer bar; regulated stirrer speed in the range of 100 to 800 rpm (decoupling may occur at higher stirrer speeds), independent for the two O2k-Chambers; automatic events sent to DatLab when the stirrer is switched on/off or when the rotation seed is changed by the experimenter. Integral component of the [[O2k-Main Unit]].[[O2k-Main Unit]].)
O2k-Main Power Cable + ('''O2k-Main Power Cable''', for connecting the main unit to the power supply.)
O2k-Peltier Temperature Control + ('''O2k-Peltier Temperature Control''': Bui … '''O2k-Peltier Temperature Control''': Built-in electronic thermostat controlling temperature for two [[O2k-chamber]]s in the range of 4 to 47 °C; ±0.002 °C (at room temperature). Continuous recording of the O2k-Copper Block temperature with DatLab. Temperature change from 20 to 30 °C within 15 min; cooling from 30 to 20 °C within 20 min. Integral component of the [[O2k-Main Unit]]. The electronic temperature control of the O2k replaced the conventional water jacket.2k replaced the conventional water jacket.)
Obesity + ('''Obesity''' is a disease resulting from … '''Obesity''' is a disease resulting from excessive accumulation of body fat. In common obesity (non-syndromic obesity) excessive body fat is due to an obesogenic lifestyle with lack of physical exercise ('couch') and caloric surplus of food consumption ('potato'), causing several comorbidities which are characterized as preventable non-communicable diseases. Persistent [[body fat excess]] associated with deficits of physical activity induces a weight-lifting effect on increasing muscle mass with decreasing mitochondrial capacity. Body fat excess, therefore, correlates with [[body mass excess]] up to a critical stage of obesogenic lifestyle-induced [[sarcopenia]], when loss of muscle mass results in further deterioration of physical performance particularly at older age.cal performance particularly at older age.)
OctGM + ('''OctGM''': [[Octanoylcarnitine]] & [[Glutamate]] & [[Malate]]. '''MitoPathway control state:''' [[FN]] '''SUIT protocols:''' [[SUIT-015]], [[SUIT-016]], [[SUIT-017]])

('''OctGM''': [[Octanoylcarnitine]] & [[Glutamate]] & [[Malate]]. '''Mit)

OctGMS + ('''OctGMS''': [[Octanoylcarnitine]] &[[Glutamate]] & [[Malate]]& [[Succinate]]. '''MitoPathway control state:''' [[FNS]] '''SUIT protocols:''' [[SUIT-016]], [[SUIT-017]])
OctM pathway control state + ('''OctM''': [[Octanoylcarnitine]] … '''OctM''': [[Octanoylcarnitine]] & [[Malate]].'''MitoPathway control state:''' F'''SUIT protocols:''' [[SUIT-002]], [[SUIT-015]], [[SUIT-016]], [[SUIT-017]]Respiratory stimulation of the [[Fatty acid oxidation pathway control state| FAO-pathway]], F, by [[fatty acid]] FA in the presence of [[malate]] M. Malate is a [[NADH Electron transfer-pathway state |type N substrate]] (N), required for the F-pathway. In the presence of [[Malate-anaplerotic pathway control state|anaplerotic pathways]] (''e.g.'', [[Malic enzyme|mitochondrial malic enzyme, mtME]]) the F-pathway capacity is overestimated, if there is an added contribution of NADH-linked respiration, F(N) (see [[SUIT-002]]). The FA concentration has to be optimized to saturate the [[Fatty acid oxidation pathway control state| FAO-pathway]], without inhibiting or uncoupling respiration. Low concentration of [[malate]], typically 0.1 mM, does not saturate the [[N-pathway]]; but saturates the [[Fatty acid oxidation pathway control state |F-pathway]]. High concentration of [[malate]], typically 2 mM, saturates the [[N-pathway]].[[N-pathway]].)
OctPGM pathway control state + ('''OctPGM''': [[Octanoylcarnitine]] … '''OctPGM''': [[Octanoylcarnitine]] & [[Pyruvate]] & [[Glutamate]] & [[Malate]].'''MitoPathway control state:''' [[FN]]'''SUIT protocols:''' [[SUIT-002]]:This substrate combination supports N-linked flux which is typically higher than FAO capacity (F/FN<1 in the OXPHOS state). In SUIT-RP1, PMOct is induced after PM(E), to evaluate any additive effect of adding Oct. In SUIT-RP2, FAO OXPHOS capacity is measured first, testing for the effect of increasing malate concentration (compare [[malate-anaplerotic pathway control state]], M alone), and pyruvate and glutamate is added to compare FAO as the background state with FN as the reference state.O as the background state with FN as the reference state.)
OctPGMS pathway control state + ('''OctPGMS''': [[Octanoylcarnitine]] … '''OctPGMS''': [[Octanoylcarnitine]] & [[Pyruvate]] & [[Glutamate]] & [[Malate]] & [[Succinate]].'''MitoPathway control state:''' [[FNS]]'''SUIT protocol:''' [[SUIT-001]], [[SUIT-002]], [[SUIT-015]]This substrate combination supports convergent electron flow to the [[Q-junction]].[[Q-junction]].)
OctPGMSGp pathway control state + ('''OctPGMSGp''': [[Octanoylcarnitine]] … '''OctPGMSGp''': [[Octanoylcarnitine]] & [[Pyruvate]] & [[Glutamate]] & [[Malate]] & [[Succinate]] & [[Glycerophosphate]].'''MitoPathway control state:''' FNSGp'''SUIT protocol:''' [[SUIT-002]]This substrate combination supports convergent electron flow to the [[Q-junction]].[[Q-junction]].)
OctPM pathway control state + ('''OctPM''': [[Octanoylcarnitine]] … '''OctPM''': [[Octanoylcarnitine]] & [[Pyruvate]] & [[Malate]].'''MitoPathway control state:''' [[FN]]'''SUIT protocol:''' [[SUIT-002]], [[SUIT-005]]This substrate combination supports N-linked flux which is typically higher than FAO capacity (F/FN<0 in the OXPHOS state). In SUIT-RP1, PMOct is induced after PM(E), to evaluate any additive effect of adding Oct. In SUIT-RP2, FAO OXPHOS capacity is measured first, testing for the effect of increasing malate concentration (compare [[malate-anaplerotic pathway control state]], M alone), and pyruvate is added to compare FAO as the background state with FN as the reference state. the background state with FN as the reference state.)
OctPMS + ('''OctPMS''': [[Octanoylcarnitine]] & [[Pyruvate]] & [[Malate]] & [[Succinate]]. '''MitoPathway control state:''' [[FNS]] '''SUIT protocol:''' [[SUIT-005]])
Octanoate + ('''Octanoate''' (octanoic acid). C8H16O2 Common name: Caprylic acid.)
Octanoylcarnitine + ('''Octanoylcarnitine''' is a medium-chain fatty acid (octanoic acid: eight-carbon saturated fatty acid) covalently linked to [[carnitine]], frequently applied as a substrate for [[fatty acid oxidation]] (FAO) in [[mitochondrial preparations]].)
Oligomycin + ('''Oligomycin''' (Omy) is an inhibitor of … '''Oligomycin''' (Omy) is an inhibitor of [[ATP synthase]] by blocking its proton channel (Fo subunit), which is necessary for oxidative phosphorylation of ADP to ATP (energy production). The inhibition of ATP synthesis also inhibits respiration. In OXPHOS analysis, Omy is used to induce a [[LEAK respiration]] state of respiration (abbreviated as ''L''(Omy) to differentiate from ''L''(n), LEAK state in the absence of ADP).L''(n), LEAK state in the absence of ADP).)