Kuznetsov 1999 Transplant Proc
| Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31: 992. |
Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Transplant Proc
Abstract: STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.1-4 To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.
• O2k-Network Lab: AT_Innsbruck_Gnaiger E, AT Innsbruck MitoCom
Labels:
Stress:Ischemia-Reperfusion; Preservation"Ischemia-Reperfusion; Preservation" is not in the list (Cell death, Cryopreservation, Ischemia-reperfusion, Permeability transition, Oxidative stress;RONS, Temperature, Hypoxia, Mitochondrial disease) of allowed values for the "Stress" property. Organism: Rat Tissue;cell: Heart Preparation: Permeabilized cells, Permeabilized tissue
Regulation: Substrate; Glucose; TCA Cycle"Substrate; Glucose; TCA Cycle" is not in the list (Aerobic glycolysis, ADP, ATP, ATP production, AMP, Calcium, Coupling efficiency;uncoupling, Cyt c, Flux control, Inhibitor, ...) of allowed values for the "Respiration and regulation" property. Coupling state: OXPHOS
HRR: Oxygraph-2k
